Articles related to NEUROPLASTICITY

Gliomas and ependymomas appear to reuse developmental hierarchies, progenitor states, and neural-circuit signals that shape tumor heterogeneity and treatment response, although many proposed therapeutic vulnerabilities remain supported primarily by preclinical evidence.

Female mice recovering from a single ketamine exposure demonstrated microglia-driven neuroplasticity through FKBP51 activation, while male mice did not. The findings identify a sex-specific biologic pathway in a preclinical model, and human relevance remains unconfirmed.

This asymmetric neuroplasticity challenges the traditional view that exercise-induced nerve adaptations are uniform, and suggests that the autonomic nervous system may fine-tune its responses based on side-specific roles in heart regulation.

Exercise-related physical activity is associated with increased brain volumes, indicating potential neuroprotective effects with effect sizes comparable across whole brain regions and AD-vulnerable areas.

Amantadine emerges as the pharmacologic agent with the most support for improving cognitive impairment after TBI, while cognitive rehabilitation interventions demonstrate substantial evidence for enhancing neurocognitive function and reducing functional limitations.

Recent research suggests that promoting neuroplasticity through various techniques may offer new treatment possibilities for patients with neurodegenerative diseases, developmental disorders, and mental health conditions.